Forkhead transcription factor FOXO subfamily is essential for reactive oxygen species-induced apoptosis
- 1 January 2008
- journal article
- Published by Elsevier in Molecular and Cellular Endocrinology
- Vol. 281 (1-2) , 47-55
- https://doi.org/10.1016/j.mce.2007.10.007
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
This publication has 39 references indexed in Scilit:
- RNA Interference–Mediated Depletion of Phosphoinositide 3-Kinase Activates Forkhead Box Class O Transcription Factors and Induces Cell Cycle Arrest and Apoptosis in Breast Carcinoma CellsCancer Research, 2006
- Phosphorylation of Serine 256 Suppresses Transactivation by FKHR (FOXO1) by Multiple MechanismsJournal of Biological Chemistry, 2002
- Forkhead transcription factor FOXO3a protects quiescent cells from oxidative stressNature, 2002
- Control of Cell Cycle Exit and Entry by Protein Kinase B-Regulated Forkhead Transcription FactorsMolecular and Cellular Biology, 2002
- 14-3-3 transits to the nucleus and participates in dynamic nucleocytoplasmic transportThe Journal of cell biology, 2002
- Reactive Oxygen Species Up-Regulates Cyclooxygenase-2, p53, and Bax mRNA Expression in Bovine Luteal CellsBiochemical and Biophysical Research Communications, 2001
- Elevated superoxide production by active H-ras enhances human lung WI-38VA-13 cell proliferation, migration and resistance to TNF-αOncogene, 2001
- Reactive oxygen species and programmed cell deathTrends in Biochemical Sciences, 1996
- Biologically relevant metal ion‐dependent hydroxyl radical generation An updateFEBS Letters, 1992
- A study of the central cavity in the bovine corpus luteumVeterinary Record, 1988