Akt Phosphorylation and Neuronal Survival after Traumatic Brain Injury in Mice
- 1 April 2002
- journal article
- Published by Elsevier in Neurobiology of Disease
- Vol. 9 (3) , 294-304
- https://doi.org/10.1006/nbdi.2002.0482
Abstract
No abstract availableKeywords
This publication has 49 references indexed in Scilit:
- Expression of Fas and Fas Ligand After Experimental Traumatic Brain Injury in the RatJournal of Cerebral Blood Flow & Metabolism, 2000
- Caspase‐3 Mediated Neuronal Death After Traumatic Brain Injury in RatsJournal of Neurochemistry, 2000
- Lithium activates the serine/threonine kinase Akt-1 and suppresses glutamate-induced inhibition of Akt-1 activity in neuronsProceedings of the National Academy of Sciences, 1999
- Akt Promotes Cell Survival by Phosphorylating and Inhibiting a Forkhead Transcription FactorCell, 1999
- Regulation of Cell Death Protease Caspase-9 by PhosphorylationScience, 1998
- Phosphatidylinositol 3-Kinase and Akt Protein Kinase Are Necessary and Sufficient for the Survival of Nerve Growth Factor-Dependent Sympathetic NeuronsJournal of Neuroscience, 1998
- Apoptosis-Suppressor Genebcl-2Expression after Traumatic Brain Injury in RatsJournal of Neuroscience, 1997
- Akt Phosphorylation of BAD Couples Survival Signals to the Cell-Intrinsic Death MachineryCell, 1997
- Inhibition of glycogen synthase kinase-3 by insulin mediated by protein kinase BNature, 1995
- Protein kinase B (c-Akt) in phosphatidylinositol-3-OH kinase signal transductionNature, 1995