Interaction and distinction of genotoxic and non-genotoxic events in carcinogenesis
- 31 December 1992
- journal article
- review article
- Published by Elsevier in Toxicology Letters
- Vol. 64-65, 597-604
- https://doi.org/10.1016/0378-4274(92)90237-e
Abstract
No abstract availableKeywords
This publication has 11 references indexed in Scilit:
- A genetic model for colorectal tumorigenesisCell, 1990
- Tissue-specific activating mutations of Ha- and Ki-ras oncogenes in skin, lung, and liver tumors induced in mice following transplacental exposure to DMBAMolecular Carcinogenesis, 1990
- Relationship between chemically induced ha‐ras mutation and transformation of BALB/c 3T3 cells: Evidence for chemical‐specific activation and cell type—specific recruitment of oncogene in transformationMolecular Carcinogenesis, 1990
- Detection of non-genotoxic carcinogens in the BALB/c 3T3 cell transformation/mutation assay systemMutagenesis, 1989
- Codon 61 Mutations in the c‐Harvey‐ras Gene in Mouse Skin Tumors Induced by 7,12‐Dimethylbenz[a]anthracene Plus Okadaic Acid Class Tumor PromotersMolecular Carcinogenesis, 1989
- Specific viral oncogenes cause differential effects on cell‐to‐cell communication, relevant to the suppression of the transformed phenotype by normal cellsMolecular Carcinogenesis, 1988
- Oncogene Activation in Chemical CarcinogenesisAdvances in Cancer Research, 1988
- Chemical structure, Salmonella mutagenicity and extent of carcinogenicity as indicators of genotoxic carcinogenesis among 222 chemicals tested in rodents by the U.S. NCI/NTPMutation Research/Genetic Toxicology, 1988
- Carcinogen-specific mutation and amplification of Ha-ras during mouse skin carcinogenesisNature, 1986
- The Clonal Evolution of Tumor Cell PopulationsScience, 1976