Erythrocyte catalase inactivation (H2O2 production) by ascorbic acid and glucose in the presence of aminotriazole: role of transition metals and relevance to diabetes

This publication has 15 references indexed in Scilit:

• Aminoguanidine: A drug proposed for prophylaxis in diabetes inhibits catalase and generates hydrogen peroxide in vitro
  Biochemical Pharmacology, 1993
• Ascorbic acid oxidation: A potential cause of the elevated severity of atherosclerosis in diabetes mellitus?
  FEBS Letters, 1992
• The effect of ascorbate supplementation on oxidative stress in the streptozotocin diabetic rat
  Free Radical Biology & Medicine, 1992
• Spirohydantoin inhibitors of aldose reductase inhibit iron- and copper-catalysed ascorbate oxidation in vitro
  Biochemical Pharmacology, 1991
• Role of Oxidative Stress in Development of Complications in Diabetes
  Diabetes, 1991
• Disturbed handling of ascorbic acid in diabetic patients with and without microangiopathy during high dose ascorbate supplementation
  Diabetologia, 1991
• Protein glycation and oxidative stress in diabetes mellitus and ageing
  Free Radical Biology & Medicine, 1991
• Reaction of ascorbate with lysine and protein under autoxidizing conditions: formation of N.epsilon.-(carboxymethyl)lysine by reaction between lysine and products of autoxidation of ascorbate
  Biochemistry, 1990
• Irreversible reaction of 3-amino-1:2:4-triazole and related inhibitors with the protein of catalase
  Biochemical Journal, 1960
• A study of the inhibition of catalase by 3-amino-1:2:4-triazole
  Biochemical Journal, 1958