Pathogenetic Role of the PML/RARα Fusion Protein in Acute Promyelocytic Leukemia
- 1 January 1996
- book chapter
- Published by Springer Nature
- Vol. 211, 269-278
- https://doi.org/10.1007/978-3-642-85232-9_27
Abstract
No abstract availableKeywords
This publication has 40 references indexed in Scilit:
- Retinoic acid regulates aberrant nuclear localization of PML-RARα in acute promyelocytic leukemia cellsCell, 1994
- Retinoid X receptor interacts with nuclear receptors in retinoic acid, thyroid hormone and vitamin D3 signallingNature, 1992
- Characterization of a Zinc Finger Gene Disrupted by the t(15;17) in Acute Promyelocytic LeukemiaScience, 1991
- The PML-RARα fusion mRNA generated by the t(15;17) translocation in acute promyelocytic leukemia encodes a functionally altered RARCell, 1991
- Chromosomal translocation t(15;17) in human acute promyelocytic leukemia fuses RARα with a novel putative transcription factor, PMLCell, 1991
- Rearrangements and aberrant expression of the retinoic acid receptor alpha gene in acute promyelocytic leukemias.The Journal of Experimental Medicine, 1990
- The t(15;17) translocation of acute promyelocytic leukaemia fuses the retinoic acid receptor α gene to a novel transcribed locusNature, 1990
- Molecular Analysis of Acute Promyelocytic Leukemia Breakpoint Cluster Region on Chromosome 17Science, 1990
- Cloning and characterization of the T(15;17) translocation breakpoint region in acute promyelocytic leukemiaGenes, Chromosomes and Cancer, 1990
- Probable in vivo induction of differentiation by retinoic acid of promyelocytes in acute promyelocytic leukaemiaBritish Journal of Haematology, 1984