Specific and sustained down-regulation of genes involved in fatty acid metabolism is not a hallmark of progression to cardiac failure in mice
- 1 June 2006
- journal article
- Published by Elsevier in Journal of Molecular and Cellular Cardiology
- Vol. 40 (6) , 838-845
- https://doi.org/10.1016/j.yjmcc.2006.03.429
Abstract
No abstract availableKeywords
This publication has 31 references indexed in Scilit:
- Cardiac fatty acid metabolism is preserved in the compensated hypertrophic rat heartBasic Research in Cardiology, 2005
- Myocardial Substrate Metabolism in the Normal and Failing HeartPhysiological Reviews, 2005
- Progressive heart failure after myocardial infarction in miceBasic Research in Cardiology, 2002
- Deactivation of peroxisome proliferator–activated receptor-α during cardiac hypertrophic growthJournal of Clinical Investigation, 2000
- Control of oxidative metabolism in volume-overloaded rat hearts: effect of propionyl-L-carnitineAmerican Journal of Physiology-Heart and Circulatory Physiology, 1997
- Fatty Acid Oxidation Enzyme Gene Expression Is Downregulated in the Failing HeartCirculation, 1996
- Sarcoplasmic Reticulum Genes Are Upregulated in Mild Cardiac Hypertrophy But Downregulated in Severe Cardiac Hypertrophy Induced by Pressure OverloadJournal of Molecular and Cellular Cardiology, 1996
- Contribution of oxidative metabolism and glycolysis to ATP production in hypertrophied heartsAmerican Journal of Physiology-Heart and Circulatory Physiology, 1994
- Alterations in cardiac gene expression during the transition from stable hypertrophy to heart failure. Marked upregulation of genes encoding extracellular matrix components.Circulation Research, 1994
- Selective changes in cardiac gene expression during compensated hypertrophy and the transition to cardiac decompensation in rats with chronic aortic banding.Circulation Research, 1993