Initiation of DNA Replication Requires the RECQL4 Protein Mutated in Rothmund-Thomson Syndrome
- 1 June 2005
- Vol. 121 (6) , 887-898
- https://doi.org/10.1016/j.cell.2005.05.015
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Defective sister-chromatid cohesion, aneuploidy and cancer predisposition in a mouse model of type II Rothmund-Thomson syndromeHuman Molecular Genetics, 2005
- RECQL4, mutated in the Rothmund–Thomson and RAPADILINO syndromes, interacts with ubiquitin ligases UBR1 and UBR2 of the N-end rule pathwayHuman Molecular Genetics, 2004
- Absence of BLM leads to accumulation of chromosomal DNA breaks during both unperturbed and disrupted S phasesThe Journal of cell biology, 2004
- Growth retardation and skin abnormalities of the Recql4-deficient mouseHuman Molecular Genetics, 2003
- Acute mutation of retinoblastoma gene function is sufficient for cell cycle re-entryNature, 2003
- S-Cdk-dependent phosphorylation of Sld2 essential for chromosomal DNA replication in budding yeastNature, 2002
- Loss of Werner syndrome protein function promotes aberrant mitotic recombinationGenes & Development, 2001
- Initiation of Eukaryotic DNA ReplicationMolecular Cell, 2000
- DNA Polymerase β and DNA Synthesis in Xenopus Oocytes and in a Nuclear ExtractScience, 1992
- DNA synthesis in a cell-free system from Xenopus eggs: Priming and elongation on single-stranded DNA in vitroCell, 1982