Ovine Lactic Acidosis: Intraruminal and Systemic2

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Abstract
Excessive grain consumption by ruminants causes a condition described as overingestion, acute indigestion, grain engorgement, and lactic acidosis, the term used in this study. In 1863, Reiset, as cited by Hungate (1966), stated that ruminants not accustomed to a grain diet often suffer acute digestive disturbances, and in many cases death, within 24 hr. after consumption of large quantities of grain. Recent research has been concerned primarily with events taking place in the rumen and the gross symptoms of the syndrome. Lactic acid is known to be increased in the rumen from 1 to 1,500 mg/100 ml (Uhart and Carrol, 1967; Walker, 1968) and in the blood from 4.5 to 90 mg/100 ml (Heuter, Shaw and Doetsch, 1956; Dunlop and Hammond, 1965) following the consumption of large amounts of grain. Furthermore, a large proportion of the lactic acid produced is D(—) lactate, which may not be metabolized systemically (Dunlop and Hammond, 1965: Ryan, 1963, 1964; Hinkson, Hoover and Poulton, 1967). Copyright © 1971. American Society of Animal Science. Copyright 1971 by American Society of Animal Science.

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