Ubiquitin and Protein Turnover in Synapse Function
- 1 September 2005
- Vol. 47 (5) , 629-632
- https://doi.org/10.1016/j.neuron.2005.07.008
Abstract
No abstract availableKeywords
This publication has 19 references indexed in Scilit:
- LIN-23-Mediated Degradation of β-Catenin Regulates the Abundance of GLR-1 Glutamate Receptors in the Ventral Nerve Cord of C. elegansNeuron, 2005
- Regulation of a DLK-1 and p38 MAP Kinase Pathway by the Ubiquitin Ligase RPM-1 Is Required for Presynaptic DevelopmentCell, 2005
- Independent Regulation of Synaptic Size and Activity by the Anaphase-Promoting ComplexCell, 2004
- The Anaphase-Promoting Complex Regulates the Abundance of GLR-1 Glutamate Receptors in the Ventral Nerve Cord of C. elegansCurrent Biology, 2004
- A proteasome-sensitive connection between PSD-95 and GluR1 endocytosisNeuropharmacology, 2004
- An SCF-like ubiquitin ligase complex that controls presynaptic differentiationNature, 2004
- Evidence for a Conserved Function in Synapse Formation Reveals Phr1 as a Candidate Gene for Respiratory Failure in Newborn MiceMolecular and Cellular Biology, 2004
- Activity level controls postsynaptic composition and signaling via the ubiquitin-proteasome systemNature Neuroscience, 2003
- Synaptic defects in ataxia mice result from a mutation in Usp14, encoding a ubiquitin-specific proteaseNature Genetics, 2002
- Ubiquitin and AP180 Regulate the Abundance of GLR-1 Glutamate Receptors at Postsynaptic Elements in C. elegansNeuron, 2002