Axonal and Nonneuronal Cell Responses to Spinal Cord Injury in Mice Lacking Glial Fibrillary Acidic Protein
- 31 December 1997
- journal article
- Published by Elsevier in Experimental Neurology
- Vol. 148 (2) , 568-576
- https://doi.org/10.1006/exnr.1997.6702
Abstract
No abstract availableKeywords
This publication has 44 references indexed in Scilit:
- Leptomeningeal cells modulate the neurite growth promoting properties of astrocytes in vitroGlia, 1997
- Topical glucocorticoids modulate the lesion interface after cerebral cortical stab wounds in adult ratsGlia, 1996
- Distinct effects of recombinant tenascin-C domains on neuronal cell adhesion, growth cone guidance, and neuronal polarityJournal of Neuroscience Research, 1996
- Mice devoid of the glial fibrillary acidic protein develop normally and are susceptible to scrapie prionsNeuron, 1995
- A novel role for myelin-associated glycoprotein as an inhibitor of axonal regenerationNeuron, 1994
- β-Amyloid of Alzheimer's Disease Induces Reactive Gliosis That Inhibits Axonal OutgrowthExperimental Neurology, 1993
- J1/tenascin is a repulsive substrate for central nervous system neuronsNeuron, 1990
- A diverse set of developmentally regulated proteoglycans is expressed in the rat central nervous systemNeuron, 1990
- Molecular specialization of astrocyte processes at nodes of Ranvier in rat optic nerve.The Journal of cell biology, 1986
- Transition between immature radial glia and mature astrocytes studied with a monoclonal antibody to vimentinDevelopmental Brain Research, 1984