Requirement of FADD for Tumor Necrosis Factor-induced Activation of Acid Sphingomyelinase
Open Access
- 1 February 1999
- journal article
- Published by Elsevier
- Vol. 274 (9) , 5267-5270
- https://doi.org/10.1074/jbc.274.9.5267
Abstract
No abstract availableKeywords
This publication has 23 references indexed in Scilit:
- FADD: Essential for Embryo Development and Signaling from Some, But Not All, Inducers of ApoptosisScience, 1998
- FLICE, A Novel FADD-Homologous ICE/CED-3–like Protease, Is Recruited to the CD95 (Fas/APO-1) Death-Inducing Signaling ComplexCell, 1996
- A Novel Cytoplasmic Domain of the p55 Tumor Necrosis Factor Receptor Initiates the Neutral Sphingomyelinase PathwayJournal of Biological Chemistry, 1996
- The TNFR2-TRAF signaling complex contains two novel proteins related to baculoviral inhibitor of apoptosis proteinsCell, 1995
- Compartmentalized Production of Ceramide at the Cell SurfacePublished by Elsevier ,1995
- Targeted mutation in the Fas gene causes hyperplasia in peripheral lymphoid organs and liverNature Genetics, 1995
- Ceramide: an endogenous regulator of apoptosis and growth suppresionImmunology Today, 1995
- The TNF receptor 1-associated protein TRADD signals cell death and NF-κB activationCell, 1995
- Functional dichotomy of neutral and acidic sphingomyelinases in tumor necrosis factor signalingCell, 1994
- A novel domain within the 55 kd TNF receptor signals cell deathCell, 1993