Herpesvirus saimiri Replaces ZAP-70 for CD3- and CD2-mediated T Cell Activation
Open Access
- 1 October 2001
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 276 (40) , 36902-36908
- https://doi.org/10.1074/jbc.m102668200
Abstract
No abstract availableKeywords
This publication has 38 references indexed in Scilit:
- Direct Binding and Activation of STAT Transcription Factors by the Herpesvirus saimiri Protein TipJournal of Biological Chemistry, 2000
- Restoration of Thymocyte Development and Function in zap-70−/− Mice by the Syk Protein Tyrosine KinaseImmunity, 1997
- Selective Activation of T Cell Kinase p56lck by Herpesvirus saimiri Protein TipPublished by Elsevier ,1996
- Immortalization of human T cells by herpesvirus saimiriImmunology Today, 1995
- The Product of the Herpesvirus saimiri Open Reading Frame 1 (Tip) Interacts with T Cell-specific Kinase p56lck in Transformed CellsPublished by Elsevier ,1995
- Immunodeficiency Diseases: Multiple roles for ZAP-70Current Biology, 1994
- Defective T cell receptor signaling and CD8+ thymic selection in humans lacking Zap-70 kinaseCell, 1994
- CD2-mediated autocrine growth of herpes virus saimiri-transformed human T lymphocytes.The Journal of Experimental Medicine, 1992
- The CD3 zeta cytoplasmic domain mediates CD2-induced T cell activation.The Journal of Experimental Medicine, 1992
- The T cell receptor/CD3 complex and CD2 stimulate the tyrosine phosphorylation of indistinguishable patterns of polypeptides in the human T leukemic cell line JurkatEuropean Journal of Immunology, 1991