A Tyrosine-Based Signal Targets H/K-ATPase to a Regulated Compartment and Is Required for the Cessation of Gastric Acid Secretion
Open Access
- 1 August 1997
- Vol. 90 (3) , 501-510
- https://doi.org/10.1016/s0092-8674(00)80510-3
Abstract
No abstract availableKeywords
This publication has 24 references indexed in Scilit:
- Gastrin-stimulated changes in Ca2+ concentration in parietal cells depends on adenosine 3′,5′-cyclic monophosphate levelsGastroenterology, 1995
- Distinct signals in the GLUT4 glucose transporter for internalization and for targeting to an insulin-responsive compartment.The Journal of cell biology, 1995
- Healing of protein losing hypertrophic gastropathy by eradication of Helicobacter pylori--is Helicobacter pylori a pathogenic factor in Menetrier's disease?Gut, 1994
- Gastric H/K+-ATPaseCellular Physiology and Biochemistry, 1994
- Menetrier's Disease and Helicobacter pyloriNew England Journal of Medicine, 1993
- Hypertrophic gastropathy resembling Ménétrier's disease in transgenic mice overexpressing transforming growth factor alpha in the stomach.Journal of Clinical Investigation, 1992
- Endocytic vesicles from renal papilla which retrieve the vasopressin-sensitive water channel do not contain a functional H+ ATPase.The Journal of cell biology, 1990
- Membrane-cytoskeleton dynamics in rat parietal cells: mobilization of actin and spectrin upon stimulation of gastric acid secretion.The Journal of cell biology, 1989
- Regulation of Transepithelial H+ Transport by Exocytosis and EndocytosisAnnual Review of Physiology, 1986
- Menetrierʼs DiseaseAnnals of Surgery, 1977