Role of ERK Activation in Growth and Erythroid Differentiation of K562 Cells
- 1 April 2001
- journal article
- Published by Elsevier in Experimental Cell Research
- Vol. 264 (2) , 193-200
- https://doi.org/10.1006/excr.2000.5124
Abstract
No abstract availableKeywords
This publication has 43 references indexed in Scilit:
- B-Raf Inhibits Programmed Cell Death Downstream of Cytochrome c Release from Mitochondria by Activating the MEK/Erk PathwayMolecular and Cellular Biology, 1999
- Requirement of Activation of JNK and p38 for Environmental Stress-Induced Erythroid Differentiation and Apoptosis and of Inhibition of ERK for ApoptosisBlood, 1999
- VEGF Prevents Apoptosis of Human Microvascular Endothelial Cells via Opposing Effects on MAPK/ERK and SAPK/JNK SignalingExperimental Cell Research, 1999
- Activation of p38 MAP Kinase and JNK But Not ERK Is Required for Erythropoietin-Induced Erythroid DifferentiationBlood, 1998
- Mitogen-activated protein kinase cascades and regulation of gene expressionCurrent Opinion in Immunology, 1996
- Opposing Effects of ERK and JNK-p38 MAP Kinases on ApoptosisScience, 1995
- The MAPK signaling cascadeThe FASEB Journal, 1995
- Specificity of receptor tyrosine kinase signaling: Transient versus sustained extracellular signal-regulated kinase activationCell, 1995
- Induction of Apoptosis by a Dominant Negative H-RAS Mutant (116Y) in K562 CellsExperimental Cell Research, 1994
- Mitogen-activated protein kinases p42mapk and p44mapk are required for fibroblast proliferation.Proceedings of the National Academy of Sciences, 1993