The dual effect of prostaglandin (PGE2) and ethanol on the natural killer cytolytic process: effector activation and NK-cell-target cell conjugate lytic inhibition.

This publication has 10 references indexed in Scilit:

• Interferon activation of "pre-spontaneous killer" (pre-SK) cells and alteration in kinetics of lysis of both "pre-SK" and active SK cells.
  The Journal of Immunology, 1980
• Suppression of Natural Cytotoxicity by Spleen Cells of Hydrocortisone-Treated Mice
  The Journal of Immunology, 1979
• Activation of Human Natural Killer Cells Cytotoxic for Human Leukemia Cells by Purified Interferon
  The Journal of Immunology, 1979
• CYTOTOXIC CELLS INDUCED DURING LYMPHOCYTIC CHORIOMENINGITIS VIRUS-INFECTION OF MICE .2. SPECIFICITIES OF THE NATURAL KILLER CELLS
  1979
• TARGET-EFFECTOR INTERACTION IN THE NATURAL KILLER CELL SYSTEM .4. MODULATION BY CYCLIC-NUCLEOTIDES
  1979
• A possible role of prostaglandins in the inhibition of natural and antibody-dependent cell-mediated cytotoxicity against tumor cells
  Cellular Immunology, 1978
• Enhancement of natural and antibody-dependent lymphocyte cytotoxicity by drugs which inhibit prostaglandin production by tumor target cells
  Cellular Immunology, 1978
• In vitro induction of cytotoxic effector cells with spontaneous killer cell specificity.
  The Journal of Experimental Medicine, 1978
• Anti-viral activity induced by culturing lymphocytes with tumor-derived or virus-transformed cells. Enhancement of human natural killer cell activity by interferon and antagonistic inhibition of susceptibility of target cells to lysis.
  The Journal of Experimental Medicine, 1978
• Subversion of host defense mechanisms by malignant tumors: an established tumor as a privileged site for bacterial growth.
  The Journal of Experimental Medicine, 1977