Modulation of Glutamate Mobility Reveals the Mechanism Underlying Slow-Rising AMPAR EPSCs and the Diffusion Coefficient in the Synaptic Cleft
- 1 June 2004
- Vol. 42 (5) , 757-771
- https://doi.org/10.1016/j.neuron.2004.04.003
Abstract
No abstract availableKeywords
This publication has 67 references indexed in Scilit:
- Maturation of EPSCs and Intrinsic Membrane Properties Enhances Precision at a Cerebellar SynapseJournal of Neuroscience, 2003
- Single synaptic vesicles fusing transiently and successively without loss of identityNature, 2003
- Fusion pore modulation as a presynaptic mechanism contributing to expression of long-term potentiationPhilosophical Transactions Of The Royal Society B-Biological Sciences, 2003
- An Evaluation of Synapse IndependenceJournal of Neuroscience, 2001
- Prolonged Synaptic Currents and Glutamate Spillover at the Parallel Fiber to Stellate Cell SynapseJournal of Neuroscience, 2000
- Postfusional regulation of cleft glutamate concentration during LTP at ‘silent synapses’Nature Neuroscience, 2000
- Transporters Buffer Synaptically Released Glutamate on a Submillisecond Time ScaleJournal of Neuroscience, 1997
- Transmitter timecourse in the synaptic cleft: its role in central synaptic functionTrends in Neurosciences, 1996
- Glutamate transporters in glial plasma membranes: Highly differentiated localizations revealed by quantitative ultrastructural immunocytochemistryNeuron, 1995
- Synaptic vesicles immunoisolated from rat cerebral cortex contain high levels of glutamateNeuron, 1989