A role for the medial region of the amygdala in mineralocorticoid-induced salt hunger.
- 1 February 1989
- journal article
- research article
- Published by American Psychological Association (APA) in Behavioral Neuroscience
- Vol. 103 (1) , 178-185
- https://doi.org/10.1037/0735-7044.103.1.178
Abstract
Damage to the medial region of the amygdala abolished aldosterone-induced salt hunger in the rat. In contrast, the salt hungers that are induced by adrenalectomy or by acute sodium depletion are left intact by the same brain damage. We suggest (a) that we have identified part of the neural circuit by which aldosterone participates in the genesis of salt hunger in the intact, sodium-depleted rat and (b) that these results provide further evidence for the hypothesis that there are separate receptive systems in the brain for the participation of aldosterone and angiotensin in the arousal of salt hunger.This publication has 3 references indexed in Scilit:
- The organization of neural inputs to the medial preoptic nucleus of the ratJournal of Comparative Neurology, 1986
- Interference with central actions of angiotensin II suppresses sodium appetiteAmerican Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 1986
- Parabrachial nuclei damage in infant rats produces residual deficits in gustatory preferences/aversions and sodium appetiteDevelopmental Psychobiology, 1983