Neurophysiology of Electroacupuncture Analgesia

This publication has 18 references indexed in Scilit:

• Monoaminergic mechanism of electroacupuncture analgesia
  Brain Research, 1981
• A combined treatment with d-amino acids and electroacupuncture produces a greater analgesia than either treatment alone; naloxone reverses these effects
  Pain, 1980
• Electroacupuncture analgesia is mediated by stereospecific opiate receptors and is reversed by antagonists of type I receptors
  Life Sciences, 1980
• Electroacupuncture Elevates Blood Cortisol Levels in Naive Horses; Sham Treatment has no Effect
  International Journal of Neuroscience, 1980
• Dynorphin-(1-13), an extraordinarily potent opioid peptide.
  Proceedings of the National Academy of Sciences, 1979
• Electroacupuncture analgesia could be mediated by at least two pain-relieving mechanisms; endorphin and non-endorphin systems
  Life Sciences, 1979
• Correlation of genetic differences in endorphin systems with analgesic effects of D-amino acids in mice
  Brain Research, 1979
• Dexamethasone partially reduces and 2% saline-treatment abolished electroacupuncture analgesia: These findings implicate pituitary endorphins
  Life Sciences, 1979
• Naloxone effects on a nociceptive response of hypophysectomized and adrenalectomized mice
  Life Sciences, 1978
• β-Endorphin and Adrenocorticotropin Are Selected Concomitantly by the Pituitary Gland
  Science, 1977