Concordant loss of MTAP and p16/CDKN2A expression in pancreatic intraepithelial neoplasia: evidence of homozygous deletion in a noninvasive precursor lesion
Open Access
- 1 July 2005
- journal article
- research article
- Published by Elsevier in Laboratory Investigation
- Vol. 18 (7) , 959-963
- https://doi.org/10.1038/modpathol.3800377
Abstract
No abstract availableKeywords
This publication has 17 references indexed in Scilit:
- Telomere Shortening Is Nearly Universal in Pancreatic Intraepithelial NeoplasiaThe American Journal of Pathology, 2002
- Chemotherapy targeting methylthioadenosine phosphorylase (MTAP) deficiency in adult T cell leukemia (ATL)Leukemia, 2002
- Pancreatic cancerCurrent Problems in Cancer, 2002
- Methylthioadenosine Phosphorylase as Target for Chemoselective Treatment of T-Cell Acute Lymphoblastic Leukemic CellsBlood Cells, Molecules, and Diseases, 2002
- Allelic Loss Is Often the First Hit in the Biallelic Inactivation of the p53 and DPC4 Genes During Pancreatic CarcinogenesisThe American Journal of Pathology, 2001
- Genetic Progression in the Pancreatic DuctsThe American Journal of Pathology, 2000
- BRCA2 Is Inactivated Late in the Development of Pancreatic Intraepithelial Neoplasia: Evidence and ImplicationsThe American Journal of Pathology, 2000
- Genomic cloning of methylthioadenosine phosphorylase: a purine metabolic enzyme deficient in multiple different cancers.Proceedings of the National Academy of Sciences, 1996
- Construction of a 2.8-megabase yeast artificial chromosome contig and cloning of the human methylthioadenosine phosphorylase gene from the tumor suppressor region on 9p21.Proceedings of the National Academy of Sciences, 1995
- Frequent somatic mutations and homozygous deletions of the p16 (MTS1) gene in pancreatic adenocarcinomaNature Genetics, 1994