Drop attacks secondary to superior canal dehiscence syndrome
- 28 June 2005
- journal article
- research article
- Published by Wolters Kluwer Health in Neurology
- Vol. 64 (12) , 2126-2128
- https://doi.org/10.1212/01.wnl.0000165953.48914.b0
Abstract
Two patients with unprovoked drop attacks were found to have dehiscence of the superior semicircular canal on CT of the temporal bone. Both had conductive hearing loss, preservation of stapedius reflex, and abnormal vestibular evoked myogenic potentials. Neither had sound- or pressure-induced nystagmus. Repair of the dehiscence in one case stopped the drop attacks, supporting a causal relationship between the dehiscence and the drop attacks.This publication has 10 references indexed in Scilit:
- Large Vestibular Evoked Myogenic Potentials in Response to Bone-Conducted Sounds in Patients with Superior Canal Dehiscence SyndromeAudiology and Neurotology, 2004
- Drop Attacks and Vertigo Secondary to a Non-Meniere Otologic CauseArchives of Neurology, 2003
- Vestibular hypersensitivity to sound (Tullio phenomenon)Neurology, 2000
- Superior canal dehiscence syndromeAmerican Journal of Otolaryngology, 2000
- Saccular and utricular inputs to sternocleidomastoid motoneurons of decerebrate catsExperimental Brain Research, 1999
- Vestibular-Evoked Myogenic Potentials in Patients with Dehiscence of the Superior Semicircular CanalActa Oto-Laryngologica, 1999
- Vestibulocollic reflexes evoked by short‐duration galvanic stimulation in manThe Journal of Physiology, 1998
- Sound- and/or Pressure-Induced Vertigo Due to Bone Dehiscence of the Superior Semicircular CanalJAMA Otolaryngology–Head & Neck Surgery, 1998
- Tapping the head activates the vestibular systemNeurology, 1995
- THE OTOLITHIC CATASTROPHE: A NEW SYNDROMEBMJ, 1936