Activation of the Tumor Metastasis Suppressor Gene, KAI1, by Etoposide Is Mediated by p53 and c-Jun Genes
- 2 August 2000
- journal article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 274 (2) , 370-376
- https://doi.org/10.1006/bbrc.2000.3139
Abstract
No abstract availableKeywords
This publication has 14 references indexed in Scilit:
- MANAGEMENT OF HORMONE REFRACTORY PROSTATE CANCER: CURRENT STANDARDS AND FUTURE PROSPECTSJournal of Urology, 1998
- The expression of the KAI1 gene, a tumor metastasis suppressor, is directly activated by p53Proceedings of the National Academy of Sciences, 1998
- Oral estramustine and oral etoposide for hormone-refractory prostate cancerUrology, 1997
- A phase ii trial of oral estramustine and oral etoposide in hormone refractory prostate cancerUrology, 1997
- KAI1 , a Metastasis Suppressor Gene for Prostate Cancer on Human Chromosome 11p11.2Science, 1995
- Therapeutic Effect of a Retroviral Wild-Type p53 Expression Vector in an Orthotopic Lung Cancer ModelJNCI Journal of the National Cancer Institute, 1994
- Inhibition of Prostate Cancer Growth by Estramustineand Etoposide: Evidence For Interaction at the Nuclear MatrixJournal of Urology, 1993
- An altered repertoire of fosjun (AP-1) at the onset of replicative senescenceExperimental Cell Research, 1992
- Characterization of the human p53 gene promoter.Molecular and Cellular Biology, 1989
- The jun proto-oncogene is positively autoregulated by its product, Jun/AP-1Published by Elsevier ,1988