Disordered eicosanoid formation in pregnancy-induced hypertension.
- 1 September 1993
- journal article
- review article
- Published by Wolters Kluwer Health in Circulation
- Vol. 88 (3) , 1324-1333
- https://doi.org/10.1161/01.cir.88.3.1324
Abstract
No abstract availableThis publication has 106 references indexed in Scilit:
- Thromboxane dominance and prostacyclin deficiency in habitual abortionThe Lancet, 1991
- The Relation of Aspirin Use during the First Trimester of Pregnancy to Congenital Cardiac DefectsNew England Journal of Medicine, 1989
- Effect of Low-Dose Aspirin on Fetal and Maternal Generation of Thromboxane by Platelets in Women at Risk for Ppregnancy-Induced HypertensionNew England Journal of Medicine, 1989
- The Use of Aspirin to Prevent Ppregnancy-Induced Hypertension and Lower the Ratio of Thromboxane A2to Prostcyclin in Relatively High Risk PregnanciesNew England Journal of Medicine, 1989
- Platelet Activation in Unstable Coronary DiseaseNew England Journal of Medicine, 1986
- Systemic production of prostacyclin and thromboxane A2 does not correlate with patency of the ductus arteriosus in very low birth weight infantsThe Journal of Pediatrics, 1986
- Increased Prostacyclin Biosynthesis in Patients with Severe Atherosclerosis and Platelet ActivationNew England Journal of Medicine, 1984
- Low prostacyclin synthetase activity of fetal rat aorta. Progressive increase after birth.Life Sciences, 1983
- PROSTACYCLIN PRODUCTION INCREASES DURING HUMAN PARTURITIONBJOG: An International Journal of Obstetrics and Gynaecology, 1981
- MATERNAL VASCULAR PROSTACYCLIN ACTIVITY IN PRE-ECLAMPSIAThe Lancet, 1980