Inhibition by Glucose of the Ethionine Induced Fatty Liver.

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Abstract
Data are presented which support the hypothesis of Recknagel and Lombardi on the genesis of fatty livers in general and especially the triglyceride accumulation after ethionine, postulating that the release from the liver into the plasma is blocked. Our data also indicate that triglyceride increase in the liver depends on both blocking of the release from, and fat mobilization into the liver from the periphery (for instance during fasting). Glucose seems to inhibit only the latter and by doing so prevents the increase of liver triglycerides by ethionine.

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