Activation of Peroxisome Proliferator-activated Receptor γ Suppresses Nuclear Factor κB-mediated Apoptosis Induced by Helicobacter pylori in Gastric Epithelial Cells

This publication has 76 references indexed in Scilit:

• Interleukin-1 polymorphisms associated with increased risk of gastric cancer
  Nature, 2000
• Tumor Necrosis Factor Alpha and Interleukin 1β Up-Regulate Gastric Mucosal Fas Antigen Expression inHelicobacter pyloriInfection
  Infection and Immunity, 2000
• Translocation of Helicobacter pylori CagA into Gastric Epithelial Cells by Type IV Secretion
  Science, 2000
• PPARγ Inhibits the Expression of c-MET in Human Gastric Cancer Cells through the Suppression of Ets
  Biochemical and Biophysical Research Communications, 1999
• The Effect of Class II Major Histocompatibility Complex Expression on Adherence of Helicobacter pylori and Induction of Apoptosis in Gastric Epithelial Cells: A Mechanism for T Helper Cell Type 1–mediated Damage
  The Journal of Experimental Medicine, 1998
• Apoptosis in Gastric Epithelial Cells Is Induced byHelicobacter pyloriand Accompanied by Increased Expression of BAK
  Biochemical and Biophysical Research Communications, 1997
• Novel Inhibitors of Cytokine-induced IκBα Phosphorylation and Endothelial Cell Adhesion Molecule Expression Show Anti-inflammatory Effects in Vivo
  Journal of Biological Chemistry, 1997
• An Essential Role for NF-κB in Preventing TNF-α-Induced Cell Death
  Science, 1996
• Alterations in cellular adhesion and apoptosis in epithelial cells overexpressing prostaglandin endoperoxide synthase 2
  Cell, 1995
• Stimulation of adipogenesis in fibroblasts by PPARγ2, a lipid-activated transcription factor
  Cell, 1994