Prenatal ethanol exposure impairs lesion-induced plasticity in a dopaminergic synapse after maturity
- 1 January 1989
- journal article
- research article
- Published by Elsevier in Neuroscience
- Vol. 29 (3) , 715-723
- https://doi.org/10.1016/0306-4522(89)90143-7
Abstract
No abstract availableThis publication has 58 references indexed in Scilit:
- Perinatal, not adult, hypothyroidism suppresses dopaminergic axon sprouting in the deafferented olfactory tubercle of adult ratJournal of Neuroscience Research, 1986
- Autoradiographic localization of D1 dopamine receptors in the rat brain with [3H]SCH 23390Brain Research, 1986
- Prenatal ethanol exposure alters the post-lesion reorganization (sprouting) of acetylcholinesterase staining in the dentate gyrus of adult ratsDevelopmental Brain Research, 1984
- Effects of maternal ethanol ingestion on cerebral neurotransmitters and cyclic-AMP in the rat offspringGeneral Pharmacology: The Vascular System, 1982
- Alterations in brain dopaminergic receptors in developing hypo- and hyperthyroid ratsNeurochemistry International, 1981
- Synaptic development in brains of rats exposed perinatally to ethanolCellular and Molecular Life Sciences, 1980
- Neuroleptic and dopamine receptors: Autoradiographic localization of [3H]spiperone in rat brainBrain Research, 1979
- Neuronal degeneration in rat brain induced by 6-hydroxydopamine; a histological and biochemical studyBrain Research, 1972
- Changes in activity and hormonal sensitivity of brain adenyl cyclase following chronic ethanol administrationCellular and Molecular Life Sciences, 1972
- THE ATTRACTIONS OF PROTEINS FOR SMALL MOLECULES AND IONSAnnals of the New York Academy of Sciences, 1949