Vinblastine-induced Phosphorylation of Bcl-2 and Bcl-XL Is Mediated by JNK and Occurs in Parallel with Inactivation of the Raf-1/MEK/ERK Cascade
Open Access
- 1 September 2000
- journal article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 275 (39) , 29980-29985
- https://doi.org/10.1074/jbc.m003776200
Abstract
No abstract availableKeywords
This publication has 22 references indexed in Scilit:
- Inhibition of c-Jun N-Terminal Kinase 2 Expression Suppresses Growth and Induces Apoptosis of Human Tumor Cells in a p53-Dependent MannerMolecular and Cellular Biology, 2000
- Translocation of SAPK/JNK to Mitochondria and Interaction with Bcl-xL in Response to DNA DamageJournal of Biological Chemistry, 2000
- TrueLeukemia, 1999
- A Functional Role for Mitochondrial Protein Kinase Cα in Bcl2 Phosphorylation and Suppression of ApoptosisJournal of Biological Chemistry, 1998
- 2-Methoxyestradiol-Induced Phosphorylation of Bcl-2: Uncoupling from JNK/SAPK ActivationBiochemical and Biophysical Research Communications, 1998
- Microtubule-interfering Agents Activate c-Jun N-terminal Kinase/Stress-activated Protein Kinase through Both Ras and Apoptosis Signal-regulating Kinase PathwaysJournal of Biological Chemistry, 1998
- Negative Modulation of Membrane Localization of the Raf-1 Protein Kinase by HyperphosphorylationJournal of Biological Chemistry, 1997
- Role of the Stress-activated/c-Jun NH2-terminal Protein Kinase Pathway in the Cellular Response to Adriamycin and Other Chemotherapeutic DrugsPublished by Elsevier ,1996
- Inactivation of Bcl-2 by phosphorylation.Proceedings of the National Academy of Sciences, 1995
- [3] Protein kinase phosphorylation site sequences and consensus specificity motifs: TabulationsPublished by Elsevier ,1991