Th1 responsiveness to nephritogenic antigens determines susceptibility to crescentic glomerulonephritis in mice
- 1 January 1997
- journal article
- Published by Elsevier in Kidney International
- Vol. 51 (1) , 94-103
- https://doi.org/10.1038/ki.1997.12
Abstract
No abstract availableKeywords
This publication has 32 references indexed in Scilit:
- Biphasic effect of interferon‐γ in murine collagen‐induced arthritisEuropean Journal of Immunology, 1995
- Th1 and Th2 CD4+ T cells in the pathogenesis of organ-specific autoimmune diseasesImmunology Today, 1995
- Effect of anti-interferon-γ monoclonal antibody treatment on the development of experimental allergic encephalomyelitis in resistant mouse strainsPublished by Elsevier ,1994
- Depletion of CD8 positive cells in nephrotoxic serum nephritis of WKY ratsKidney International, 1992
- Prevention of diabetes in NOD mice treated with antibody to murine IFNγJournal of Autoimmunity, 1991
- PARTICIPATION OF CELL-MEDIATED IMMUNITY IN DEPOSITION OF FIBRIN IN GLOMERULONEPHRITISThe Lancet, 1988
- Prevention of type II collagen-induced arthritis by in vivo treatment with anti-L3T4.The Journal of Experimental Medicine, 1985
- Monoclonal antibody to murine gamma interferon inhibits lymphokine-induced antiviral and macrophage tumoricidal activities.The Journal of Experimental Medicine, 1984
- Mononuclear-cell subsets in human idiopathic crescentic glomerulonephritis (ICGN): Analysis in tissue sections with monoclonal antibodiesJournal of Clinical Immunology, 1984
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976