Ganglioside AGF2 promotes task-specific recovery and attenuates the cholinergic hypofunction induced by AF64A
- 1 September 1990
- journal article
- research article
- Published by Elsevier in Brain Research
- Vol. 527 (2) , 299-307
- https://doi.org/10.1016/0006-8993(90)91150-f
Abstract
No abstract availableKeywords
This publication has 45 references indexed in Scilit:
- Hemicholinium-3 prevents the working memory impairments and the cholinergic hypofunction induced by ethylcholine aziridinium ion (AF64A)Brain Research, 1989
- AF64A-induced working memory impairment: behavioral, neurochemical and histological correlatesBrain Research, 1988
- GM1 ganglioside enhances synaptosomal resistance to chemically induced damageNeuroscience Letters, 1988
- AF64A (ethylcholine aziridinium ion), a cholinergic neurotoxin, selectively impairs working memory in a multiple component T-maze taskBrain Research, 1987
- Retrograde changes in the nucleus basalis of the rat, caused by cortical damage, are prevented by exogenous ganglioside GM1Brain Research, 1986
- Effects of ganglioside treatment in rats with a lesion of the cholinergic forebrain nucleiBrain Research, 1985
- Monosialoganglioside internal ester stimulates the dopaminergic reinnervation of the striatum after unilateral hemitransection in ratInternational Journal of Developmental Neuroscience, 1984
- Memory and the septo-hippocampal cholinergic system in the ratPsychopharmacology, 1983
- The Cholinergic Hypothesis of Geriatric Memory DysfunctionScience, 1982
- A rapid and sensitive method for the quantitation of microgram quantities of protein utilizing the principle of protein-dye bindingAnalytical Biochemistry, 1976