Host cell reactivation of plasmids containing oxidative DNA lesions is defective in Cockayne syndrome but normal in UV-sensitive syndrome fibroblasts
- 29 August 2005
- journal article
- research article
- Published by Elsevier in DNA Repair
- Vol. 5 (1) , 13-22
- https://doi.org/10.1016/j.dnarep.2005.06.017
Abstract
No abstract availableKeywords
This publication has 39 references indexed in Scilit:
- UV-sensitive syndromeMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 2005
- Transcription and DNA adducts: what happens when the message gets cut off?DNA Repair, 2004
- Effect of 8-oxoguanine on transcription elongation by T7 RNA polymerase and mammalian RNA polymerase IIDNA Repair, 2004
- Effect of Thymine Glycol on Transcription Elongation by T7 RNA Polymerase and Mammalian RNA Polymerase IIJournal of Biological Chemistry, 2001
- Reduced global genomic repair of ultraviolet light-induced cyclobutane pyrimidine dimers in simian virus 40-transformed human cellsMolecular Carcinogenesis, 2000
- RETRACTED: Transcription-Coupled Repair of 8-oxoGuanine: Requirement for XPG, TFIIH, and CSB and Implications for Cockayne SyndromeCell, 2000
- UVs Syndrome: Establishment and Characterization of Fibroblastic Cell Lines Transformed with Simian Virus 40 DNAJournal of Investigative Dermatology, 2000
- Simian virus 40 (SV40) large T antigen-dependent amplification of an Epstein-Barr virus-SV40 hybrid shuttle vector integrated into the human HeLa cell genomeJournal of General Virology, 1992
- Induction of the Escherichia coli lactose operon selectively increases repair of its transcribed DNA strandNature, 1989
- Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogeneNature, 1984