Little or no induction of hyperglycemia by 2-deoxy-d-glucose in hereditary blind microphthalmic rats
- 1 January 1988
- journal article
- research article
- Published by Elsevier in Life Sciences
- Vol. 43 (20) , 1575-1582
- https://doi.org/10.1016/0024-3205(88)90528-0
Abstract
No abstract availableThis publication has 23 references indexed in Scilit:
- Loss of a circadian adrenal corticosterone rhythm following suprachiasmatic lesions in the ratPublished by Elsevier ,2003
- Temporary Suppression of the Hyperglycemic Response to 2-Deoxy-d-Glucose by BlindingChronobiology International, 1988
- Circadian rhythms in sleep-wakefulness and wheel-running activity in a congenitally anophthalmic rat mutantPhysiology & Behavior, 1987
- Role of SCN in Daily Rhythms of Plasma Glucose, FFA, Insulin and GlucagonChronobiology International, 1987
- Bilateral Lesions Involving the Suprachiasmatic Nucleus Suppress Hyperglycemia due to Peripheral Administration of 2-Deoxy-D-GlucoseHormone and Metabolic Research, 1986
- Bilateral lesions of the SCN abolish lipolytic and hyperphagic responses to 2DGPhysiology & Behavior, 1984
- Quantitative analysis of the lateral geniculate nucleus in the mutant microphthalmic ratExperimental Neurology, 1983
- Ontogeny of the vasopressinergic neurons of the suprachiasmatic nucleus and their extrahypothalamic projections in the rat brain—presence of a sex difference in the lateral septumBrain Research, 1981
- Morphogenesis of the hereditary microphthalmia in a new strain of ratJournal of Morphology, 1981
- Der Einfluß des Augenlichtes auf StoffwechselvorgängeJournal Of Neural Transmission-Parkinsons Disease and Dementia Section, 1967