Inhibition of the Epithelial Na+ Channel by Interaction of Nedd4 with a PY Motif Deleted in Liddle's Syndrome

This publication has 34 references indexed in Scilit:

• Liddle disease caused by a missense mutation of beta subunit of the epithelial sodium channel gene.
  Journal of Clinical Investigation, 1996
• A de novo missense mutation of the beta subunit of the epithelial sodium channel causes hypertension and Liddle syndrome, identifying a proline-rich segment critical for regulation of channel activity.
  Proceedings of the National Academy of Sciences, 1995
• Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channel
  Cell, 1995
• Hypertension caused by a truncated epithelial sodium channel γ subunit: genetic heterogeneity of Liddle syndrome
  Nature Genetics, 1995
• A mutation in the epithelial sodium channel causing Liddle disease increases channel activity in the Xenopus laevis oocyte expression system.
  Proceedings of the National Academy of Sciences, 1995
• Liddle's syndrome: Heritable human hypertension caused by mutations in the β subunit of the epithelial sodium channel
  Cell, 1994
• Amiloride-sensitive epithelial Na+ channel is made of three homologous subunits
  Nature, 1994
• Expression cloning of an epithelial amiloride‐sensitive Na⁺ channel
  FEBS Letters, 1993
• Epithelial sodium channel related to proteins involved in neurodegeneration
  Nature, 1993
• THE LDL RECEPTOR LOCUS IN FAMILIAL HYPERCHOLESTEROLEMIA: Mutational Analysis of a Membrane Protein
  Annual Review of Genetics, 1990