Cascading towards vascular disorder gene therapy
- 1 June 1995
- journal article
- editorial
- Published by Springer Nature in Nature Medicine
- Vol. 1 (6) , 513-515
- https://doi.org/10.1038/nm0695-513
Abstract
No abstract availableKeywords
This publication has 12 references indexed in Scilit:
- Inhibition of cellular ras prevents smooth muscle cell proliferation after vascular injury in vivoNature Medicine, 1995
- Thyrotropin-Induced Mitogenesis Is Ras Dependent but Appears To Bypass the Raf-Dependent Cytoplasmic Kinase CascadeMolecular and Cellular Biology, 1995
- The MEK Kinase Activity of the Catalytic Domain of RAF-1 Is Regulated Independently of Ras Binding in T CellsPublished by Elsevier ,1995
- The ins and outs of Raf kinasesTrends in Biochemical Sciences, 1994
- Gene Therapy for Vascular Smooth Muscle Cell Proliferation After Arterial InjuryScience, 1994
- The pathogenesis of atherosclerosis: a perspective for the 1990sNature, 1993
- Serum-, TPA-, and Ras-induced expression from Ap-1/Ets-driven promoters requires Raf-1 kinase.Genes & Development, 1992
- Inhibition of phosphatidylinositol 4-phosphate kinase by heparin. A possible mechanism for the antiproliferative effects of heparinBiochemical Journal, 1992
- Inhibitors of Angiotensin-Converting Enzyme Prevent Myointimal Proliferation After Vascular InjuryScience, 1989
- Inhibition of NIH 3T3 cell proliferation by a mutant ras protein with preferential affinity for GDP.Molecular and Cellular Biology, 1988