The tumor suppressor p53 modifies mutational processes in a human lymphoblastoid cell line
- 1 January 1997
- journal article
- Published by Elsevier in Mutation Research - Fundamental and Molecular Mechanisms of Mutagenesis
- Vol. 373 (1) , 87-97
- https://doi.org/10.1016/s0027-5107(96)00193-5
Abstract
No abstract availableKeywords
This publication has 48 references indexed in Scilit:
- Separate domains of p21 involved in the inhibition of Cdk kinase and PCNANature, 1995
- The dominating effect of mutant p53Nature Genetics, 1995
- p53 binds single-stranded DNA ends through the C-terminal domain and internal DNA segments via the middle domainNucleic Acids Research, 1995
- p53-Dependent apoptosis in the absence of transcriptional activation of p53-target genesNature, 1994
- p53 binds single-stranded DNA ends and catalyzes DNA renaturation and strand transfer.Proceedings of the National Academy of Sciences, 1994
- Inverted DNA repeats: a source of eukaryotic genomic instability.Molecular and Cellular Biology, 1993
- Different cytotoxic and mutagenic responses induced by X-rays in two human lymphoblastoid cell lines derived from a single donorMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1993
- A comparison of induced mutation at homologous alleles of the tk locus in human cells. II. Molecular analysis of mutantsMutation Research - Fundamental and Molecular Mechanisms of Mutagenesis, 1992
- Mice deficient for p53 are developmentally normal but susceptible to spontaneous tumoursNature, 1992
- Presence of a Potent Transcription Activating Sequence in the p53 ProteinScience, 1990