STOP knockout and NMDA NR1 hypomorphic mice exhibit deficits in sensorimotor gating
- 8 September 2005
- journal article
- research article
- Published by Elsevier in Behavioural Brain Research
- Vol. 163 (2) , 257-264
- https://doi.org/10.1016/j.bbr.2005.05.012
Abstract
No abstract availableKeywords
This publication has 35 references indexed in Scilit:
- Disturbance of sensory filtering in dementia with Lewy bodies: comparison with Parkinson's disease dementia and Alzheimer's diseaseJournal of Neurology, Neurosurgery & Psychiatry, 2005
- Placebo-Controlled Evaluation of Four Novel Compounds for the Treatment of Schizophrenia and Schizoaffective DisorderAmerican Journal of Psychiatry, 2004
- The suppression of brain cold-stable microtubules in mice induces synaptic defects associated with neuroleptic-sensitive behavioral disordersGenes & Development, 2002
- Is the GABA cell a final common pathway for the etiology and treatment of schizophrenia and bipolar disorder?Current Opinion in Psychiatry, 2002
- STOP Proteins are Responsible for the High Degree of Microtubule Stabilization Observed in Neuronal CellsThe Journal of cell biology, 1998
- On the Plausibility of “The Neurodevelopmental Hypothesis” of SchizophreniaNeuropsychopharmacology, 1996
- Targeted disruption of NMDA receptor 1 gene abolishes NMDA response and results in neonatal deathPublished by Elsevier ,1994
- Dopamine receptors and the dopamine hypothesis of schizophreniaSynapse, 1987
- Microtubule reassembly from nucleating fragments during the regrowth of amputated neurites.The Journal of cell biology, 1986
- Disorders of attention and perception in early schizophreniaPsychology and Psychotherapy: Theory, Research and Practice, 1961