The BH3 domain is required for caspase-independent cell death induced by Bax and oligomycin
- 11 April 2000
- journal article
- Published by Springer Nature in Cell Death & Differentiation
- Vol. 7 (4) , 338-349
- https://doi.org/10.1038/sj.cdd.4400659
Abstract
Cell death and differentiation is a monthly research journal focused on the exciting field of programmed cell death and apoptosis. It provides a single accessible source of information for both scientists and clinicians, keeping them up-to-date with advances in the field. It encompasses programmed cell death, cell death induced by toxic agents, differentiation and the interrelation of these with cell proliferation.Keywords
This publication has 34 references indexed in Scilit:
- Molecular characterization of mitochondrial apoptosis-inducing factorNature, 1999
- The Bcl-2 Protein Family: Arbiters of Cell SurvivalScience, 1998
- Mitochondria and ApoptosisScience, 1998
- BCL-2 FAMILY: Regulators of Cell DeathAnnual Review of Immunology, 1998
- Mitochondrial cytochrome c release in apoptosis occurs upstream of DEVD-specific caspase activation and independently of mitochondrial transmembrane depolarizationThe EMBO Journal, 1998
- Movement of Bax from the Cytosol to Mitochondria during ApoptosisThe Journal of cell biology, 1997
- Prevention of Apoptosis by Bcl-2: Release of Cytochrome c from Mitochondria BlockedScience, 1997
- The Release of Cytochrome c from Mitochondria: A Primary Site for Bcl-2 Regulation of ApoptosisScience, 1997
- Bcl-2 inhibits the mitochondrial release of an apoptogenic protease.The Journal of Experimental Medicine, 1996
- Induction of Apoptotic Program in Cell-Free Extracts: Requirement for dATP and Cytochrome cPublished by Elsevier ,1996