Inhibition of gastric H+,K+-ATPase and acid secretion by SCH 28080, a substituted pyridyl(1,2a)imidazole.
Open Access
- 1 February 1987
- journal article
- research article
- Published by Elsevier in Journal of Biological Chemistry
- Vol. 262 (5) , 2077-2084
- https://doi.org/10.1016/s0021-9258(18)61620-5
Abstract
No abstract availableThis publication has 32 references indexed in Scilit:
- Cell biology: Is there a common design for cell membrane channels?Nature, 1986
- The mechanism for inhibition of gastric (H+ + K+)-ATPase by omeprazoleBiochimica et Biophysica Acta (BBA) - Biomembranes, 1985
- Evidence for acid-induced transformation of omeprazole into an active inhibitor of (H+ + K+)-ATPase within the parietal cellBiochimica et Biophysica Acta (BBA) - Biomembranes, 1984
- Kinetic properties of the KCl transport at the secreting apical membrane of the oxyntic cellThe Journal of Membrane Biology, 1983
- Vanadate binding to the gastric hydrogen(1+)-transporting, potassium(1+)-stimulated ATPase and inhibition of the enzyme's catalytic and transport activitiesBiochemistry, 1983
- ATP/ADP exchange activity of gastric (H+ + K+)-ATPaseBiochimica et Biophysica Acta (BBA) - Biomembranes, 1982
- Quantitation of hydrogen ion and potential gradients in gastric plasma membrane vesiclesBiochemistry, 1978
- Cation transport by gastric H+∶K+ ATPaseThe Journal of Membrane Biology, 1977
- A Method for Preparing Isolated Glands from the Rabbit Gastric MucosaActa Physiologica Scandinavica, 1976
- On the reversibility of binding of cardiotonic steroids to a partially purified (Na + K)-activated adenosinetriphosphatase from beef brainBiochemical and Biophysical Research Communications, 1970