Disruption of synaptosomal calcium channel function by Lambert-Eaton myasthenic immunoglobulin is serum-dependent
- 25 December 1992
- journal article
- Published by Elsevier in Brain Research
- Vol. 599 (2) , 317-323
- https://doi.org/10.1016/0006-8993(92)90407-z
Abstract
No abstract availableKeywords
This publication has 29 references indexed in Scilit:
- Specificity of Lambert-Eaton Myasthenic Syndrome immunoglobulin for nerve terminal calcium channelsBrain Research, 1992
- Serum and plasma from patients with Lambert-Eaton myasthenic syndrome reduce depolarization-dependent uptake of 45Ca2+ into rat cortical synaptosomesBrain Research, 1991
- Further Characterization of Phasic Calcium Influx in Rat Cerebrocortical Synaptosomes: Inferences Regarding Calcium Channel Type(s) in Nerve EndingsJournal of Neurochemistry, 1989
- Immune mechanisms in acute and chronic inflammatory polyneuropathiesJournal of Neuroimmunology, 1988
- Voltage-Dependent Ca2+ Channels in Small Cell Carcinomas Are Blocked by Autoantibodies from Patients with Lambert-Eaton Myasthenic SyndromeAnnals of the New York Academy of Sciences, 1988
- Bay K 8644 stimulation of calcium entry and endogenous dopamine release in rat striatal synaptosomes antagonized by nimodipineBrain Research, 1986
- The Biochemistry of Opsonization: Central Role of the Reactive Thiolester of the Third Component of ComplementThe Journal of Infectious Diseases, 1984
- The biochemistry of complementNature, 1978
- Alternative pathway of complement: demonstration and characterization of initiating factor and its properdin-independent function.The Journal of Experimental Medicine, 1976
- The opsonic fragment of the third component of human complement (C3).The Journal of Experimental Medicine, 1975