Release of Endogenous ATP from Rat Caudal Artery

Abstract
Electrical field stimulation of the rat caudal artery (0.5-ms pulses at 8 Hz for 3 min) results in the release of norepinephrine (quantified by HPLC-electrochemical detection) and adenyl purines including ATP, ADP and AMP (quantified by HPLC-fluorescence detection). The amount of ATP released from the tissue exceeded the amount of norepinephrine. Because postjunctional α1-adrenoceptor stimulation with methoxamine also causes release of ATP, both neuronal and extraneuronal sites may contribute to the overflow of ATP. Results with the α1-adrenoceptor antagonist prazosin lend support to this notion. Prazosin (10–6M) completely blocked the release of ATP by methoxamine but only partially reduced the release of ATP by field stimulation.

This publication has 0 references indexed in Scilit: