Down regulation of DJ-1 enhances cell death by oxidative stress, ER stress, and proteasome inhibition
Top Cited Papers
- 1 December 2003
- journal article
- research article
- Published by Elsevier in Biochemical and Biophysical Research Communications
- Vol. 312 (4) , 1342-1348
- https://doi.org/10.1016/j.bbrc.2003.11.056
Abstract
No abstract availableKeywords
Funding Information
- Ministry of Education, Culture, Sports, Science and Technology
- Ministry of Health, Labour and Welfare
This publication has 20 references indexed in Scilit:
- L166P Mutant DJ-1, Causative for Recessive Parkinson's Disease, Is Degraded through the Ubiquitin-Proteasome SystemJournal of Biological Chemistry, 2003
- The role of pathogenic DJ‐1 mutations in Parkinson's diseaseAnnals of Neurology, 2003
- An Integrated Stress Response Regulates Amino Acid Metabolism and Resistance to Oxidative StressPublished by Elsevier ,2003
- Oxidative stress in Parkinson's diseaseAnnals of Neurology, 2003
- Mutations in the DJ-1 Gene Associated with Autosomal Recessive Early-Onset ParkinsonismScience, 2003
- Parkin Protects against the Toxicity Associated with Mutant α-SynucleinNeuron, 2002
- DJ-1 Positively Regulates the Androgen Receptor by Impairing the Binding of PIASxα to the ReceptorJournal of Biological Chemistry, 2001
- Parkin Suppresses Unfolded Protein Stress-induced Cell Death through Its E3 Ubiquitin-protein Ligase ActivityJournal of Biological Chemistry, 2000
- Iron in the basal ganglia in Parkinson's diseaseBrain, 1999
- DJ-1, a Novel Oncogene Which Transforms Mouse NIH3T3 Cells in Cooperation withrasBiochemical and Biophysical Research Communications, 1997