Involvement of caspase-1 proteases in hypoxic brain injury. Effects of their inhibitors in developing neurons
- 28 December 1999
- journal article
- Published by Elsevier in Neuroscience
- Vol. 95 (4) , 1157-1165
- https://doi.org/10.1016/s0306-4522(99)00501-1
Abstract
No abstract availableKeywords
This publication has 41 references indexed in Scilit:
- Transient hypoxia may lead to neuronal proliferation in the developing mammalian brain: from apoptosis to cell cycle completionNeuroscience, 1999
- Sequential activation of activator protein-1-related transcription factors and JNK protein kinases may contribute to apoptotic death induced by transient hypoxia in developing brain neuronsMolecular Brain Research, 1998
- Induction of Caspase-3-Like Protease May Mediate Delayed Neuronal Death in the Hippocampus after Transient Cerebral IschemiaJournal of Neuroscience, 1998
- Caspase inhibitor affords neuroprotection with delayed administration in a rat model of neonatal hypoxic-ischemic brain injury.Journal of Clinical Investigation, 1998
- Hypoxia/reoxygenation induces apoptosis through biphasic induction of protein synthesis in cultured rat brain neuronsBrain Research, 1998
- Caspases: the executioners of apoptosisBiochemical Journal, 1997
- Activation of a CrmA-insensitive, p35-sensitive Pathway in Ionizing Radiation-induced ApoptosisPublished by Elsevier ,1997
- Mammalian cell death proteases: A family of highly conserved aspartate specific cysteine proteasesJournal of Cellular Biochemistry, 1997
- The cell-death machineCurrent Biology, 1996
- Mechanisms of delayed cell death following hypoxic-ischemic injury in the immature rat: evidence for apoptosis during selective neuronal lossMolecular Brain Research, 1995