A novel accessory role of neutrophils in concanavalin A-induced hepatitis
- 31 January 2005
- journal article
- Published by Elsevier in Cellular Immunology
- Vol. 233 (1) , 23-29
- https://doi.org/10.1016/j.cellimm.2005.03.003
Abstract
No abstract availableKeywords
This publication has 20 references indexed in Scilit:
- CCL3/MIP‐1α is pro‐inflammatory in murine T cell‐mediated hepatitis by recruiting CCR1‐expressing CD4+ T cells to the liverEuropean Journal of Immunology, 2004
- Mouse bone marrow contains large numbers of functionally competent neutrophilsJournal of Leukocyte Biology, 2003
- Macrophage inflammatory protein-2 induced by TNF-α plays a pivotal role in concanavalin A-induced liver injury in miceJournal of Hepatology, 2001
- Concanavalin A-induced Hepatitis in Mice is Prevented by Interleukin (IL)-10 and Exacerbated by Endogenous IL-10 DeficiencyAutoimmunity, 1999
- Involvement of Fas/Fas ligand system-mediated apoptosis in the development of concanavalin A-induced hepatitisEuropean Journal of Immunology, 1998
- In vivo evidence for a functional role of both tumor necrosis factor (TNF) receptors and transmembrane TNF in experimental hepatitisEuropean Journal of Immunology, 1997
- Interferon gamma plays a critical role in T cell-dependent liver injury in mice initiated by concanavalin AGastroenterology, 1996
- Interleukin‐12 production by human polymorphonuclear leukocytesEuropean Journal of Immunology, 1995
- Regression of an established tumor genetically modified to release granulocyte colony-stimulating factor requires granulocyte-T cell cooperation and T cell-produced interferon gamma.The Journal of Experimental Medicine, 1993
- A rapid method for the isolation of functional thymus‐derived murine lymphocytesEuropean Journal of Immunology, 1973