Essential thrombocythemias without V617F JAK2 mutation are clonal hematopoietic stem cell disorders
Open Access
- 6 April 2006
- journal article
- letter
- Published by Springer Nature in Leukemia
- Vol. 20 (6) , 1181-1183
- https://doi.org/10.1038/sj.leu.2404214
Abstract
No abstract availableThis publication has 10 references indexed in Scilit:
- X-inactivation–based clonality analysis and quantitative JAK2V617F assessment reveal a strong association between clonality and JAK2V617F in PV but not ET/MMM, and identifies a subset of JAK2V617F-negative ET and MMM patients with clonal hematopoiesisBlood, 2006
- Detection of JAK2 V617F as a first intention diagnostic test for erythrocytosisLeukemia, 2005
- Definition of subtypes of essential thrombocythaemia and relation to polycythaemia vera based on JAK2 V617F mutation status: a prospective studyThe Lancet, 2005
- Clinical implications of the JAK2 V617F mutation in essential thrombocythemiaLeukemia, 2005
- On the molecular origins of the chronic myeloproliferative disorders: it all makes senseBlood, 2005
- A Large Proportion of Patients With a Diagnosis of Essential Thrombocythemia Do Not Have a Clonal Disorder and May Be at Lower Risk of Thrombotic ComplicationsBlood, 1999
- X-Chromosome inactivation in healthy females: incidence of excessive lyonization with age and comparison of assays involving DNA methylation and transcript polymorphismsClinical Chemistry, 1998
- Clonality Analysis of Hematopoiesis in Essential Thrombocythemia: Advantages of Studying T Lymphocytes and PlateletsBlood, 1997
- Use of Restriction Fragment Length Polymorphisms to Determine the Clonal Origin of Human TumorsScience, 1985