Cyclosporin A Induces a Biphasic Increase in KCl-Induced Calcium Influx in GH3 Pituitary Cells

This publication has 19 references indexed in Scilit:

• Subunit interaction sites in voltage-dependent Ca2+ channels: role in channel function
  Trends in Neurosciences, 1998
• The Molecular Basis of Voltage-gated Ca 2+ Channel Diversity: Is It Time for T?
  The Journal of Membrane Biology, 1998
• Mechanism of Ca 2+ -dependent Inactivation of L-type Ca 2+ Channels in GH 3 Cells: Direct Evidence Against Dephosphorylation by Calcineurin
  The Journal of Membrane Biology, 1997
• Structure and Regulation of L-Type Calcium Channels: A Current Assessment of the Properties and Roles of Channel Subunits
  Trends in Cardiovascular Medicine, 1996
• Protein-tyrosine Kinases Activate while Protein-tyrosine Phosphatases Inhibit L-type Calcium Channel Activity in Pituitary GH3 Cells
  Published by Elsevier ,1996
• Activation of protein kinase C reduces L-type calcium channel activity of GH3 pituitary cells
  American Journal of Physiology-Cell Physiology, 1992
• Calcium-dependent inactivation of the dihydropyridine-sensitive calcium channels in GH3 cells.
  The Journal of general physiology, 1988
• Voltage-activated calcium channels that must be phosphorylated to respond to membrane depolarization.
  Proceedings of the National Academy of Sciences, 1987
• An enzymatic mechanism for calcium current inactivation in dialysed Helix neurones.
  The Journal of Physiology, 1986
• Establishment of Clonal Strains of Rat Pituitary Tumor Cells That Secrete Growth Hormone^1,2
  Endocrinology, 1968