Splenic lipofuscinosis in mice

Abstract
Autopsy examination of young adult mice revealed a characteristic pigmentation of the anterior splenic pole occurring in a high proportion (8-34%) of 3 mouse strains and 2 sublines. Histological studies identified the pigment as lipofuscin and EM provided supporting evidence. Lipofuscin may represent non-metabolizable debris from cellular breakdown associated with lysosomal activity.