Ouabain Is a Potent Promoter of Growth and Activator of ERK1/2 in Ouabain-resistant Rat Renal Epithelial Cells

This publication has 47 references indexed in Scilit:

• Src-mediated Inter-receptor Cross-talk between the Na+/K+-ATPase and the Epidermal Growth Factor Receptor Relays the Signal from Ouabain to Mitogen-activated Protein Kinases
  Journal of Biological Chemistry, 2002
• Digoxin
  Progress in Cardiovascular Diseases, 2002
• Involvement of Src and Epidermal Growth Factor Receptor in the Signal-transducing Function of Na+/K+-ATPase
  Journal of Biological Chemistry, 2000
• Chromosome Cohesion, Condensation, and Separation
  Annual Review of Biochemistry, 2000
• Compensatory renal growth in uninephrectomized adult mice is growth hormone dependent
  Kidney International, 1999
• Angiotensin II Stimulates Secretion of Endogenous Ouabain From Bovine Adrenocortical Cells via Angiotensin Type 2 Receptors
  Hypertension, 1997
• Partial Inhibition of Na+/K+-ATPase by Ouabain Induces the Ca2+-dependent Expressions of Early-response Genes in Cardiac Myocytes
  Journal of Biological Chemistry, 1996
• Long-term ouabain administration produces hypertension in rats.
  Hypertension, 1993
• Ouabain Resistance Conferred by Expression of the cDNA for a Murine Na ⁺ , K ⁺ -ATPase α Subunit
  Science, 1987
• Cellular basis for the species differences in sensitivity to cardiac glycosides (digitalis)
  Journal of Cellular Physiology, 1986