Dopamine D3 receptor and schizophrenia: A widened scope for the immune hypothesis

This publication has 43 references indexed in Scilit:

• Dopamine by itself activates either D2, D3 or D1/D5 dopaminergic receptors in normal human T-cells and triggers the selective secretion of either IL-10, TNFα or both
  Journal of Neuroimmunology, 2005
• Reductions in neurotrophin receptor mRNAs in the prefrontal cortex of patients with schizophrenia
  Molecular Psychiatry, 2005
• Mechanisms of Relapse Prevention in Schizophrenia
  Pharmacopsychiatry, 2004
• Serum IL‐1β, sIL‐2R, IL‐6, IL‐8 and TNF‐α in schizophrenic patients, relation with symptomatology and responsiveness to risperidone treatment
  Mediators of Inflammation, 2000
• Differential effects of Th1 and Th2 derived cytokines on NGF synthesis by mouse astrocytes
  FEBS Letters, 1996
• The Effects of Haloperidol on Dopamine Receptor Gene Expression
  Experimental Neurology, 1994
• The effect of haloperidol on D2 dopamine receptor subtype mRNA levels in the brain
  FEBS Letters, 1994
• Molecular cloning and characterization of a novel dopamine receptor (D3) as a target for neuroleptics
  Nature, 1990
• Dopamine receptor subtype imbalance in schizophrenia
  Life Sciences, 1987
• Antipsychotic drug doses and neuroleptic/dopamine receptors
  Nature, 1976