Rescue of developing spinal motoneurons from programmed cell death by the GABAA agonist muscimol acts by blockade of neuromuscular activity and increased intramuscular nerve branching
- 31 March 2003
- journal article
- Published by Elsevier in Molecular and Cellular Neuroscience
- Vol. 22 (3) , 331-343
- https://doi.org/10.1016/s1044-7431(02)00020-9
Abstract
No abstract availableKeywords
This publication has 46 references indexed in Scilit:
- Target-Derived Cardiotrophin-1 and Insulin-like Growth Factor-I Promote Neurite Growth and Survival of Developing Oculomotor NeuronsMolecular and Cellular Neuroscience, 2002
- Synaptic Assembly of the Brain in the Absence of Neurotransmitter SecretionScience, 2000
- Retinal Ganglion Cells Lose Trophic Responsiveness after AxotomyNeuron, 1999
- Schwann cell apoptosis at developing neuromuscular junctions is regulated by glial growth factorNature, 1996
- Motoneurons deprived of trophic support in vitro require new gene expression to undergo programmed cell deathJournal of Neurobiology, 1994
- Interactions between spinal cord stimulation and activity blockade in the regulation of synaptogenesis and motoneuron survival in the chick embryoJournal of Neurobiology, 1993
- The relationship of intramuscular nerve branching and synaptogenesis to motoneuron survivalJournal of Neurobiology, 1992
- Naturally occurring and induced neuronal death in the chick embryo in vivo requires protein and RNA synthesis: Evidence for the role of cell death genesDevelopmental Biology, 1990
- Cell death of motoneurons in the chick embryo spinal cord. IV. Evidence that a functional neuromuscular interaction is involved in the regulation of naturally occurring cell death and the stabilization of synapsesJournal of Comparative Neurology, 1979
- Neuromuscular blockade increases motoneurone survival during normal cell death in the chick embryoNature, 1978