Sur1 Knockout Mice
Open Access
- 1 March 2000
- journal article
- Published by Elsevier
- Vol. 275 (13) , 9270-9277
- https://doi.org/10.1074/jbc.275.13.9270
Abstract
No abstract availableKeywords
This publication has 37 references indexed in Scilit:
- A New ER Trafficking Signal Regulates the Subunit Stoichiometry of Plasma Membrane KATP ChannelsNeuron, 1999
- Tissue-Specific Knockout of the Insulin Receptor in Pancreatic β Cells Creates an Insulin Secretory Defect Similar to that in Type 2 DiabetesCell, 1999
- Glucose action `beyond ionic events' in the pancreatic β cellTrends in Pharmacological Sciences, 1998
- Paternal mutation of the sulfonylurea receptor (SUR1) gene and maternal loss of 11p15 imprinted genes lead to persistent hyperinsulinism in focal adenomatous hyperplasia.Journal of Clinical Investigation, 1998
- The sulphonylurea receptor SUR1 regulates ATP‐sensitive mouse Kir6.2 K+ channels linked to the green fluorescent protein in human embryonic kidney cells (HEK 293)The Journal of Physiology, 1998
- Association and Stoichiometry of KATP Channel SubunitsPublished by Elsevier ,1997
- Truncation of Kir6.2 produces ATP-sensitive K+ channels in the absence of the sulphonylurea receptorNature, 1997
- Familial Persistent Hyperinsulinemic Hypoglycemia of Infancy and Mutations in the Sulfonylurea ReceptorNew England Journal of Medicine, 1997
- Mechanisms by which glucose can control insulin release independently from its action on adenosine triphosphate-sensitive K+ channels in mouse B cells.Journal of Clinical Investigation, 1993
- Evidence that glucose can control insulin release independently from its action on ATP-sensitive K+ channels in mouse B cells.Journal of Clinical Investigation, 1992