Transamination Reaction in Normal and B6-Deficient Hamsters

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Abstract
The transamination rate of the heart muscle of B6-deficient hamsters was found to be 30 to 40% lower than that of control ad libitum-fed or pair-fed animals. The in vitro addition of pyridoxal alone, pyridoxal and ATP, and pyridoxal phosphate was without effect. Injections of desoxypyridoxine into experimental animals and the in vitro addition of the drug alone or together with ATP failed to produce any significant changes. Deficient hamsters exhibited succinoxidase activities comparable to those of control animals.

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