Regulation of the Epithelial Sodium Channel by N4WBP5A, a Novel Nedd4/Nedd4-2-interacting Protein
Open Access
- 1 August 2002
- journal article
- Published by Elsevier
- Vol. 277 (33) , 29406-29416
- https://doi.org/10.1074/jbc.m203018200
Abstract
No abstract availableKeywords
This publication has 34 references indexed in Scilit:
- Epithelial Sodium Channel and the Control of Sodium Balance: Interaction Between Genetic and Environmental FactorsAnnual Review of Physiology, 2002
- Structure and Regulation of Amiloride-Sensitive Sodium ChannelsAnnual Review of Physiology, 2000
- Mutations causing Liddle syndrome reduce sodium-dependent downregulation of the epithelial sodium channel in the Xenopus oocyte expression system.Journal of Clinical Investigation, 1998
- Epithelial sodium channels: function, structure, and regulationPhysiological Reviews, 1997
- Cell surface expression of the epithelial Na channel and a mutant causing Liddle syndrome: A quantitative approachProceedings of the National Academy of Sciences, 1996
- A de novo missense mutation of the beta subunit of the epithelial sodium channel causes hypertension and Liddle syndrome, identifying a proline-rich segment critical for regulation of channel activity.Proceedings of the National Academy of Sciences, 1995
- Mechanism by which Liddle's syndrome mutations increase activity of a human epithelial Na+ channelCell, 1995
- Hypertension caused by a truncated epithelial sodium channel γ subunit: genetic heterogeneity of Liddle syndromeNature Genetics, 1995
- A mutation in the epithelial sodium channel causing Liddle disease increases channel activity in the Xenopus laevis oocyte expression system.Proceedings of the National Academy of Sciences, 1995
- Liddle's syndrome: Heritable human hypertension caused by mutations in the β subunit of the epithelial sodium channelCell, 1994